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Lymphokine receptors: a structural diversity. The genes expressed for this chemokine family are all assigned to a single chromosome 5q. NK Type 1 cells are identified according to their level of expression of the IL-2Ralpha chain. Unlike normal immunocytes that are capable of activation by antigens on the cell surface, lymphokine activation does not necessarily involve T-cell recognition of antigen-bearing cells. These receptors are all assigned to the type 1 – interleukin-2 receptor alpha (IL2RA) – and the type 5 – interleukin-7 receptor alpha (IL7RA) – gene families respectively. An opposing function to cell activation is activation, which occurs when cells are sensitized with a ligand for an antigen-specific receptor, so that cells can be engaged in antibody-dependent cellular cytotoxicity. NK cells are activated in response to the binding of NK cell ligands to their receptors. This inhibits another type of cell that is not yet identified. With respect to species differences, when studies were done on mice, particularly humanized mice and human NK cells, the function and differentiation of these cells appeared to be somewhat different than the function of human NK cells. Pro-inflammatory cytokines normally activate monocytes, NK cells and mast cells, and all of these cells normally mediate protection against bacterial, viral and parasitic infections. However, when inflammation is prolonged, other cells produce an anti-inflammatory cytokine IL-10 to inhibit the pro-inflammatory cells. T-cells normally lyse infected or transformed cells when other signals such as CD4 and CD8 are activated, but the activation of NK cells is also mediated by proteins called KIRs. Antibodies of the immunoglobulin class appear to have an effect on the ability of NK cells to kill infected cells. NK cells have recently been found to be inhibited by certain cytokines, such as TGF-beta and IL-10. These cytokines can be produced by several cell types, such as T-cells, monocytes, dendritic cells, epithelial cells, endothelial cells, adipocytes, some fibroblasts, thymus cells, and some hematopoietic precursors in the bone marrow. These cytokines bind to specific receptors (TGF-beta receptor I, type-1 IL-10 receptor, and the IL-2Ralpha chain).

This transfer is directed by the expression of Ly49 family cell surface receptors. The normal

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